CLINICAL CONUNDRUM IN THE TREATMENT OF EPTIFIBATIDE-INDUCED THROMBOCYTOPENIA FOLLOWING PERCUTANEOUS CORONARY INTERVENTION
نویسندگان
چکیده
منابع مشابه
Cerebral Hemorrhage Complicating Heparin-induced Thrombocytopenia after Percutaneous Coronary Intervention: A Clinical Dilemma of Medical Treatment
Correspondence To the Editor: Little is known about the management of acute myocardial infarction in patient with heparin‑induced thrombocytopenia (HIT), especially after the primary percutaneous coronary intervention (PCI). We present a case of intracerebral hemorrhage (ICH) in the left temporal lobe after PCI. An 81‑year‑old man was admitted to our hospital who complained of severe chest pain...
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Background—Platelet glycoprotein (GP) IIb/IIIa antagonists prevent the composite end point of death or myocardial infarction (MI) in patients with acute coronary syndromes. There is uncertainty about whether this effect is confined to patients who have percutaneous coronary interventions (PCIs) and whether PCIs further prevent death or MI in patients already treated with GP IIb/IIIa antagonists...
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BACKGROUND Platelet glycoprotein (GP) IIb/IIIa antagonists prevent the composite end point of death or myocardial infarction (MI) in patients with acute coronary syndromes. There is uncertainty about whether this effect is confined to patients who have percutaneous coronary interventions (PCIs) and whether PCIs further prevent death or MI in patients already treated with GP IIb/IIIa antagonists...
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One of the major dilemmas facing physicians is what diagnostic and therapeutic approaches should be recommended to those stable coronary artery disease patients whose symptoms are adequately controlled on medical therapy. This study sought to assess the evidence-based data relating to whether: 1) all patients with significant coronary lesions (i.e., ischemia-producing) should undergo percutaneo...
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Heparin Induced Thrombocytopenia (HIT) is a rare disorder, which manifests as acute onset thrombocytopenia within five to fifteen days after exposure to heparin. The milder version of the disease may manifest as asymptomatic thrombocytopenia, which resolves with cessation of heparin. The more severe form presents with arterial and venous thrombi, profound thrombocytopenia and is associated with...
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ژورنال
عنوان ژورنال: Journal of the American College of Cardiology
سال: 2015
ISSN: 0735-1097
DOI: 10.1016/s0735-1097(15)60700-8